ArticleGlucose, insulin, and open field responses to immobilization in nonobese diabetic (NOD) mice
References (46)
Tests for emotionality in rats and mice: A review
Anim. Behav.
(1973)- et al.
Influence of catecholaminergic and serotonergic receptor antagonists on the hyperglycaemic response to the neuroglucopaenic agent, 2-deoxy-d-glucose
Neuropharmacology
(1991) - et al.
Effects of various environmental stress paradigms and adrenalectomy on the expression of IDDM in the nonobese diabetic (NOD) mouse
J. Autoimmun.
(1993) - et al.
Depletion of hypothalamic norepinephrine and serotonin enhances the dexamethasone negative feedback on adrenocortical secretion
Psychoendocrinology
(1991) - et al.
Glucocorticoids in the nonobese diabetic (NOD) mouse: Basal serum levels, effect of endocrine manipulation and immobilization stress
Life Sci.
(1992) - et al.
Sex steroids, glucocorticoids, stress and autoimmunity
J. Steroid Biochem. Mol. Biol.
(1991) - et al.
Central serotonergic responses and behavioural adaptation to repeated immobilisation: The effect of the corticosterone synthesis inhibitor metyrapone
Eur. J. Pharmacol.
(1985) - et al.
Female rats are more vulnerable than males in an animal model of depression: The possible role of serotonin
Brain Res.
(1986) - et al.
Exaggerated peripheral responses to catecholamines contributes to stress-induced hyperglycemia in the ob/ob mouse
Pharmacol. Biochem. Behav.
(1987) Control of spontaneous glucose intolerance, hyperinsulinemia, and islet hyperplasia in nonobese C3H.SW male mice by Y-linked locus and adrenal gland
Metabolism
(1988)
Enhanced metabolism of arachidonic acid by macrophages from nonobese diabetic (NOD) mice
Clin. Immunol. Immunopathol.
Centrally mediated drug-induced hyperglycemia in mice
Neuropharmacology
Group size, cage shelf level, and emotionality in non-obese diabetic mice: Impact on onset and incidence of IDDM
Psychosom. Med.
Pharmacological characterization of the hyperglycemia induced by alpha-2 adrenoceptor agonists
J. Pharmacol. Exp. Ther.
Corticosterone response to the serotonergic agonist D-fenfluramine may be independent from corticotropin-releasing factor (CRF)
Neurosci. Lett.
Psychophysiological aspects of the management of diabetes mellitus
Int. J. Ment. Health
Effects of 2-deoxyglucose on carbohydrate metabolism: Review of the literature and studies in the rat
Metabolism
Effect of stress on blood glucose in IDDM
Diabetes Care
Behavior, stress, and lymphocyte recirculation
Mechanisms involved in the hyperglycemic response induced by clonidine and other alpha-2 adrenoceptor agonists
J. Pharmacol. Exp. Ther.
Induction and therapy of autoimmune diabetes in the non-obese diabetic (NOD/Lt) mouse by a 65-kDa heat shock protein
Anterior pituitary gland
The glycemic effects of sympathomimetics in stressed mice
Res. Commun. Chem. Pathol. Pharmacol.
Cited by (20)
Monocyte-derived dendritic cells in bipolar disorder
2006, Biological PsychiatryCitation Excerpt :A similar imbalance, a defective DC development in favor of the macrophages, has been found in the NOD mouse model for autoimmune thyroiditis and insulitis (Nikolic et al 2005). Interestingly, NOD mice also show behavioral and hormonal abnormalities, such as a high locomotor activity in open field tests and an intrinsically high corticosterone response to repeated constraint stress, which have been taken as evidence for an altered emotional behavior of the mice (Amrani et al 1994; Durant et al 1998). With regard to the previously posed question of whether the here-described DC aberrancies might be of relevance for the psychiatric phenotype, it is also worth noting that DC and brain microglia are strongly related (Aloisi et al 1999; Ulvestad et al 1994).
Increased astrocyte reactivity in the hippocampus of murine models of type 1 diabetes: The nonobese diabetic (NOD) and streptozotocin-treated mice
2002, Brain ResearchCitation Excerpt :These neuropeptide changes may reflect a potentially high sensitivity to stress due to diabetes acting as an endogenous metabolic stressor [60]. In this regard, diabetic humans and both NOD mice and STZ-diabetic animals appear to be hypersensitive to stress [2–4,10,14,62]. Our objective was to study astrocyte reactivity in the NOD hippocampus during the prediabetic and diabetic stage and in mice rendered diabetic by STZ treatment.