Experimental Corynebacterium pseudotuberculosis infection in lambs: kinetics of bacterial dissemination and inflammation
References (35)
- et al.
Experimental infection of sheep with Salmonella typhimurium
Res. Vet. Sci.
(1976) - et al.
Immunity to intracellular bacteria
Vet. Immunol. Immunopathol.
(1982) CD8+ T lymphocytes in intracellular microbial infections
Immunol. Today
(1988)Sheep leukocyte molecules: a review of their distribution, structure and possible function
Vet. Immunol. Immunopathol.
(1988)- et al.
Epithelioid cell granuloma induction in the guinea pig by haptenated Mycobacterium leprae
Cell. Immunol.
(1987) - et al.
An abattoir survey of the prevalence of caseous lymphadenitis in feral goats in Western Australia
Aust. Vet. J.
(1985) Aspects of pathogenesis in a mouse model of infection by Corynebacterium pseudotuberculosis
Aust. J. Exp. Biol. Med. Sci.
(1986)Frequency and consequence of caseous lymphadenitis in sheep and lambs slaughtered at a Western Australian abattoir
Am. J. Vet. Res.
(1986)Pathogenesis of caseous lymphadenitis in sheep and goats
Aust. Vet. J.
(1986)- et al.
Prevalence and distribution of caseous lymphadenitis in feral goats
Aust. Vet. J.
(1986)
Epididymitis in rams. Studies on pathogenesis
Cornell Vet.
Caseous lymphadenitis of goats and sheep: a review
Vet. Bull.
Interleukines et inflammation
Pathol. Biol.
Copper and ceruloplasmin (Cp) concentrations during the acute inflammatory process in the rat
Agents Actions
Pathogenesis of pulmonary tuberculosis
Am. Rev. Respir. Dis.
Cytokines: Interleukin-1 and tumour necrosis factor (cachectin)
Recruitment of 99mm-Technetium or 111-Indium labelled polymorphonuclear leucocytes in experimentally induced pyogranulomas in lambs
J. Leuk. Biol.
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Clinico-pathological responses and PCR detection of Corynebacterium pseudotuberculosis and its immunogenic mycolic acid extract in the vital organs of goats
2019, Microbial PathogenesisCitation Excerpt :The rumen motility of C. pseudotuberculosis inoculated group was found to manifest some degree of reduction. Dissemination of CLA infection may also emerge from the transfer of the bacteria through the vascular or lymphatic system to other organs and this may lead to the formation of lesions at other organs within the host [6,22]. This could be associated with the size and number of the abscesses found within the mediastinal lymph nodes which may exert some sort of pressure on the adjacent ruminal structure as they grow in size thereby interfering with normal rumination as previously reported [29].
Diseases of the Hematopoietic and Hemolymphatic Systems
2019, Large Animal Internal MedicineProbing the heat shock response of Corynebacterium pseudotuberculosis: The major virulence factor, phospholipase D, is downregulated at 43 °C
2007, Research in MicrobiologyCitation Excerpt :Knowledge regarding the mechanisms by which disease is then established has, in general, been based on experimental studies in which bacteria have been administered by subcutaneous inoculation. Pepin et al. [28,29] have demonstrated that experimental infection in sheep is typically associated with a transient temperature increase, localised inflammation and abscess formation at the site of infection [28]. It is likely that bacteria replicate extracellularly at the site of infection prior to lymph node localisation.
Salmonella abortusovis, strain Rv6, a new vaccinal vehicle for small ruminants
1998, Veterinary MicrobiologyAcute phase proteins and transformed cells
1997, International Review of Cytology