Summary
Postreplication repair is defective in fibroblasts from all the xeroderma pigmentosum (XP) complementation groups with the exception of group E, the defect being most pronounced in the excision-proficient XP variants. It is normal in cells from patients with a variety of other disorders. During postreplication repair pyrimidine dimer sites become associated with daughter strands in both normal and XP cells.
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Lehmann, A.R., Kirk-Bell, S., Arlett, C.F. (1977). Postreplication Repair in Human Fibroblasts. In: Castellani, A. (eds) Research in Photobiology. Springer, Boston, MA. https://doi.org/10.1007/978-1-4613-4160-4_30
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DOI: https://doi.org/10.1007/978-1-4613-4160-4_30
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