Abstract
The role of intracranial pressure gradients in the development of neurologic deficits after cerebral infarction has not been evaluated adequately, partly because of technical problems with measurements of regional intracranial pressure (ICP). The presumed cause of post-infarction ICP gradients is ischemic cerebral edema; as a preliminary to a study of edema and its modification by treatment, ICP was measured in cats after occlusion of one middle cerebral artery (MCA).
Supported in part by Research Grant NS-3364 from the National Institutes of Health, U.S. Public Health Service.
Recipient of an International Postdoctoral Research Fellowship from the National Institutes of Health, U.S. Public Health Service.
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References
O’Brien, M. D., Waltz, A. G.: Transorbital approach for experimental occlusion of middle cerebral artery without craniectomy. (In press.)
Brock, M., Beck, J., Markakis, E., Dietz, H.: Intracranial pressure gradients associated with experimental cerebral embolism. Stroke 3, 123–130 (1972).
Halsey, J. H., Jr., Capra, N. F.: The course of experimental cerebral infarction — the development of increased intracranial pressure. Stroke 3, 268–278 (1972).
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© 1972 Springer-Verlag Berlin · Heidelberg
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O’Brien, M.D., Waltz, A.G. (1972). Intracranial Pressure Changes during Experimental Cerebral Infarction. In: Brock, M., Dietz, H. (eds) Intracranial Pressure. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-642-65486-2_18
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DOI: https://doi.org/10.1007/978-3-642-65486-2_18
Publisher Name: Springer, Berlin, Heidelberg
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