Biochemical and Biophysical Research Communications
Volume 182, Issue 2, 31 January 1992, Pages 727-732
The effect of interleukin-4 on the macrophage respiratory burst is species dependent
References (23)
- et al.
Immunol. Today
(1988) Blood
(1991)- et al.
Cell. Immunol
(1989) - et al.
Biochem. Biophys. Res. Commun
(1987) - et al.
Ann. Rev. Immunol
(1987) - et al.
Immunology
(1990) - et al.
J. Leukocyte Biol
(1991) - et al.
J. Immunol
(1990) - et al.
J. Immunol
(1989) - et al.
Immunology
(1991)
Cited by (14)
Alternative activation of macrophages by IL-4 enhances the proteolytic capacity of their phagosomes through synergistic mechanisms
2011, BloodCitation Excerpt :NOX2 is a major antimicrobial effector of the macrophage's phagosome. Conflicting observations regarding the effect of IL-4 treatment on the extracellular secretion of superoxide anions by macrophages have been reported.9,11,32,40-43 Here, we demonstrate that IL-4, but not IL-13, reliably diminishes the NOX2-mediated respiratory burst in the phagosome in both BMMØs and PMØs.
Use of bone marrow-derived macrophages to model murine innate immune responses
2006, Journal of Immunological MethodsLipopolysaccharide-induced priming of the human neutrophil is not associated with a change in phosphotyrosine phosphatase activity
1999, International Journal of Biochemistry and Cell BiologyAntimicrobial mechanisms of activated macrophages and their induction by cytokines
1996, Advances in Structural BiologyEffector functions of activated macrophages against parasites
1993, Current Opinion in Immunology
Copyright © 1992 Published by Elsevier Inc.