Chapter 106 - Sepsis

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  • Role of neuroendocrine modulation and biochemistry in the sepsis in Piaractus mesopotamicus

    2020, General and Comparative Endocrinology
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    In this study, cortisol was the key mediator for hyperglycemia after inoculation of A. hydrophila, considering that at 6 and 9 HPI there was a decrease in serum cortisol and glycaemia levels compared to control group. This immediate increase of glycaemia followed by its reduction is similar to the observed in sepsis of dogs (Boller and Otto, 2009), cats (Parsons et al., 2009), crustaceans (Lorenzon et al., 1997), and humans (Branco et al., 2007). Although glycaemia increase is a characteristic of clinical infections (Cely et al., 2004), experimentally Harikrishnan and Balasundaram (2003) observed a reduction of glycemic levels in Cyprinus carpio 10 days after inoculation of A. hydrophila.

  • Increased concentrations of C-reactive protein but not high-mobility group box 1 in dogs with naturally occurring sepsis

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    Sepsis, defined as systemic inflammatory response syndrome (SIRS) caused by an infection (Levy et al., 2003) is associated with high mortality rates worldwide in both humans and animals (Bone et al., 1992; Brun-Buisson et al., 1996; Angus et al., 2001; King et al., 2001; Harrison et al., 2006; Blanco et al., 2008; Boller and Otto, 2009; Esper and Martin, 2009).

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