Emotion socialization within the family environment and adolescent depression

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Abstract

This review evaluates research addressing the association between parent–child emotional interactions and the development and maintenance of depression in adolescence, with a focus on studies using observational research methods that assess parental responses to children and adolescents' emotional displays. We argue that parental socialization behaviors in response to different emotions expressed by youths may have distinct associations with depressive outcomes. In particular, parental behaviors that reinforce depressive behavior, reciprocate aggression, and fail to positively reinforce positive behavior have each been associated with youth depression. This review identifies a need for more observational research, including prospective, longitudinal studies, to better understand these behaviors, elucidate the directionality of influence between parental socialization behaviors and youth depression, and more clearly identify protective parental socialization behaviors. However, the use of existing findings to inform family-based interventions may improve prevention and treatment efforts directed at youth depression.

Highlights

► Parental socialization of emotion is associated with adolescent depressive outcomes. ► Socialization patterns with different emotions show different associations with depression. ► Depression is associated with parental reinforcement of sadness and reciprocation of anger. ► Lack of parental reinforcement of positive behavior is also associated with youth depression.

Introduction

Epidemiological research has shown that the onset of major depressive disorder (MDD) commonly occurs in adolescence, with an estimated 20–28% of young people experiencing an episode of MDD by age 19 (Hankin et al., 1998, Kessler et al., 2005, Lewinsohn et al., 1998). Episodes of adolescent depression have been associated with concurrent and future psychosocial dysfunction (Fergusson and Woodward, 2002, Lewinsohn et al., 2003, Lewinsohn et al., 1998) and with high rates of recurrence (Hankin et al., 1998, Lewinsohn et al., 1999). Despite advances in intervention development, a significant proportion of depressed adolescents fail to respond to available evidence-based treatments (Gladstone & Beardslee, 2009), and preventive interventions evidence relatively modest efficacy (Horowitz and Garber, 2006, Merry et al., 2004). Given the substantial humanitarian and economic costs of depression (World Health Organization, 2008), a better understanding of the risk and protective factors that contribute to the onset and maintenance of depression is required in order to develop better interventions to prevent and treat this disorder.

This review critically examines research into the influence of parenting behaviors on risk for depression, which is an important and potentially modifiable component of risk that could be targeted in preventive and treatment interventions for adolescent depression. We begin with an evaluation and integration of research into one aspect of parental socialization of emotion, namely parents' response to expressions of sadness, anger, and positive emotions by their children. This discussion leads to the proposal of a preliminary model linking patterns of parental responses to different emotions with youth depression. Directions for future research and clinical implications arising from this model conclude the review.

Section snippets

Parental socialization of emotion

Numerous family factors have been investigated in relation to their influence on the development and maintenance of youth depression. These factors can be considered on a continuum from “macro” to “micro” aspects of the family environment, ranging from (i) broad, contextual family stressors such as parental psychopathology, marital conflict, and economic disadvantage, to (ii) parenting style, defined as the broad approach to parenting, including attitudes and beliefs that shape the emotional

A preliminary model

Based on the research reviewed above, we propose a preliminary model summarizing the most consistently reported associations between parental responses to their child's expression of dysphoric, angry and positive emotions, and youth depression (see Fig. 1). Different interaction patterns involving the three youth emotions were identified, suggesting that a number of different mechanisms relating to the socialization of emotions may be associated with youth depression. In particular, the

Directions for future research

Overall, the evidence base reviewed above is limited and more observational research is required to better understand the different mechanisms by which parental responses to their children's emotional behaviors may influence youth depression. Furthermore, given the mixed results obtained in some studies, it is recommended that potential moderators of the relationship between parental socialization behaviors and youth depression be explored. Such moderators could include psychological or

Clinical implications

The research considered in this review highlights the importance of including parenting components in preventive and treatment interventions for adolescent depression. Most notably, the evidence suggests that particular parental socialization behaviors are associated with depression in young people, and so educating parents about adaptive and maladaptive ways of responding to their child's expression of emotion may protect against negative outcomes.

Parents have commonly been involved in

Conclusion

The way in which parents socialize the expression of emotion in their children has been found to be associated with youth depression, in there is some evidence to suggest that some PSE behaviors may prospectively influence the onset of disorder. This review argues that PSE behaviors in response to different emotions expressed by children and adolescents may have different associations with depressive outcomes. In particular, the existing evidence base suggests that parental reinforcement of

Acknowledgments

This research was supported by grants from the Colonial Foundation, the National Health and Medical Research Council (NHMRC Program Grant 350241) and the Australian Research Council (Discovery Grants DP0878136 and DP1092637).

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