Journal of Biological Chemistry
Signal TransductionH2O2 oxidation of cysteine residues in c-Jun N-terminal kinase 2 (JNK2) contributes to redox regulation in human articular chondrocytes
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This work was supported by National Institutes of Health Grants R37 AR049003 and RO1 AG044034 (to R. F. L.), F31 AG032796 (to E. A. E.), R33 CA126659 (to L. B. P.), and R33 CA177461 (to C. M. F.). The authors declare that they have no conflicts of interest with the contents of this article. The content is solely the responsibility of the authors and does not necessarily represent the official views of the National Institutes of Health.
This article contains Figs. S1–S7 and Table S1.
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Present address: National Institutes of Health Undiagnosed Diseases Program, National Institutes of Health, Bethesda, MD 20892.
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Present address: Dept. of Biochemistry and Molecular Genetics, University of Colorado, Anschutz Medical Campus, Aurora, CO 80045.
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Present address: Dept. of Nano Science and Engineering, South Dakota School of Mines and Technology, Rapid City, SD 57701.