New Observations Letters
Opsoclonus-Myoclonus- Ataxia Syndrome (OMAS) Associated with SARS-CoV-2 Infection: Post-Infectious Neurological Complication with Benign Prognosis
Authors:
- Enrique Urrea-MendozaEmail Enrique Urrea-Mendoza
- Kimberly Okafor
- Senthuran Ravindran
- John Absher
- Varun Chaubal
- Fredy J. Revilla
Abstract
The novel coronavirus SARS‐CoV‐2 (severe acute respiratory syndrome coronavirus-2) is the cause of the COVID‐19 pandemic [5]. SARS-Cov-2 demonstrates partial resemblance to SARS-CoV and MERS-CoV in phylogenetic analysis, clinical manifestations, and pathological findings [6, 7]. Reports emerging from China have described ataxia as a neurological symptom of the SARS‐CoV‐2 infection [5]. Opsoclonus consists of back-to-back multidirectional conjugate saccades without an inter-saccadic interval [8]. Myoclonus is defined as a sudden, brief, “shock‐like”, nonepileptic involuntary movement [9], which has been described as a symptom of SARS-CoV-2 infection [10]. Opsoclonus-Myoclonus-Ataxia syndrome (OMAS) associated COVID-19 infection has been reported recently [11, 12].
- Year: 2021
- Volume: 11
- Page/Article: 7
- DOI: 10.5334/tohm.580
- Submitted on 28 Oct 2020
- Accepted on 21 Jan 2021
- Published on 10 Feb 2021
- Peer Reviewed